Eric Verdin
- Espace de travail: Japon, Osaka, CELL GRAND CLINIC
- Specialist at the clinic: Dr. Eric Verdin (President, Buck Institute for Research on Aging) is cited in the clinic’s NMN/NAD+ content for skepticism about intravenous NAD+ therapy.
- In a discussion with physician Peter Attia on The Drive (episode #359), he explained that NAD+ is too large to efficiently enter cells and is broken down into nicotinamide before it can be used.
- He recommends oral precursors such as NMN as a more practical approach to restoring NAD+ biology.
As a specialist at the clinic, Dr. Eric Verdin (President of the Buck Institute for Research on Aging) is referenced in the clinic’s NAD+ discussion for why intravenous NAD+ may not reach cells effectively. In a conversation with Peter Attia on The Drive (episode #359), he highlighted NAD+’s molecular size and described breakdown into nicotinamide before cellular use. The clinic’s longevity messaging therefore leans toward oral precursors like NMN as a more workable option.
Services du médecin
Où en sommes-nous ?
Qualifications
Education
Work experience
- Buck Institute for Research on Aging — President (role stated in the clinic materials).
Clinical focus and techniques
Dr. Verdin’s contribution in the clinic materials is preventive and mechanistic: evaluating how NAD+ biology may be supported through precursors rather than direct NAD+ infusion.
- NAD+ vs NMN cellular uptake considerations (molecular-size rationale described in the clinic’s NAD+ content).
- Recommendation for oral NMN precursors over intravenous NAD+ (as described in the clinic’s cited discussion).
Professional societies and certifications
Academic output
Courses and advanced training
Awards and recognition
The clinic uses Dr. Verdin’s mechanistic critique to frame its longevity-related messaging around NAD+ restoration strategies. For patients, the practical takeaway presented in the clinic materials is that NMN is positioned as a more cell-uptake-friendly precursor than NAD+ for supporting NAD+ biology.
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